BDNF Facilitates L-LTP Maintenance in the Absence of Protein Synthesis through PKMζ

Open Access

Peer-reviewed

Research Article

Fan Mei,

Affiliations School of Basic Medical Sciences, Peking University Health Science Center, Beijing, China, Program in Developmental Neurobiology, Eunice Kennedy Shriver, National Institute of Child Health and Human Development, Bethesda, Maryland, United States of America
⨯
Guhan Nagappan,

Affiliations Program in Developmental Neurobiology, Eunice Kennedy Shriver, National Institute of Child Health and Human Development, Bethesda, Maryland, United States of America, GlaxoSmithKline, R&D China, Pudong, Shanghai, China
⨯
Yang Ke,

Affiliation School of Basic Medical Sciences, Peking University Health Science Center, Beijing, China
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Todd C. Sacktor,

Affiliation The Robert F. Furchgott Center of Neural and Behavioural Science, Departments of Physiology and Pharmacology and Neurology, State University of New York (SUNY) Downstate Medical Center, Brooklyn, New York, United States of America
⨯
Bai Lu

* E-mail: bai.b.lu@gsk.com

Affiliations Program in Developmental Neurobiology, Eunice Kennedy Shriver, National Institute of Child Health and Human Development, Bethesda, Maryland, United States of America, GlaxoSmithKline, R&D China, Pudong, Shanghai, China
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Protein synthesis
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Hippocampus
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Neurons
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Tetanus
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Long-term potentiation
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MAPK signaling cascades
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Synthesis phase
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Long term memory
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