LYL1 Degradation by the Proteasome Is Directed by a N-Terminal PEST Rich Site in a Phosphorylation-Independent Manner

Open Access

Peer-reviewed

Research Article

Georgi L. Lukov,

Affiliations Department of Pediatrics, Baylor College of Medicine, Houston, Texas, United States of America, Stem Cells and Regenerative Medicine Center, Baylor College of Medicine, Houston, Texas, United States of America
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Margaret A. Goodell

* E-mail: goodell@bcm.edu

Affiliations Department of Pediatrics, Baylor College of Medicine, Houston, Texas, United States of America, Stem Cells and Regenerative Medicine Center, Baylor College of Medicine, Houston, Texas, United States of America
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Phosphorylation
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Proteasomes
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Proline
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293T cells
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Immunoprecipitation
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Transfection
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T cells
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Transcription factors
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