BRCA1 Interacts with Smad3 and Regulates Smad3-Mediated TGF-β Signaling during Oxidative Stress Responses

Open Access

Peer-reviewed

Research Article

Huchun Li,

Affiliation Division of Experimental Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts, United States of America
⨯
Masayuki Sekine,

Affiliation Division of Experimental Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts, United States of America
⨯
Seyha Seng,

Affiliation Division of Experimental Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts, United States of America
⨯
Shalom Avraham,

Affiliation Division of Experimental Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts, United States of America
⨯
Hava Karsenty Avraham

* E-mail: havraham@bidmc.harvard.edu

Affiliation Division of Experimental Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts, United States of America
⨯

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Genetic causes of cancer
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Immunoprecipitation
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Oxidative stress
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Luciferase
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Transfection
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SMAD signaling
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Monoclonal antibodies
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Breast cancer
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