Retinoic acid alleviates the reduction of Akt and Bad phosphorylation and regulates Bcl-2 family protein interactions in animal models of ischemic stroke

Ju-Bin Kang; Phil-Ok Koh

doi:10.1371/journal.pone.0303213

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Open Access

Peer-reviewed

Research Article

Retinoic acid alleviates the reduction of Akt and Bad phosphorylation and regulates Bcl-2 family protein interactions in animal models of ischemic stroke

Ju-Bin Kang,

Roles Data curation, Methodology, Software, Visualization, Writing – original draft

Affiliation Department of Anatomy, College of Veterinary Medicine, Research Institute of Life Science, Gyeongsang National University, Jinju, South Korea
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Phil-Ok Koh

Roles Conceptualization, Funding acquisition, Supervision, Writing – original draft, Writing – review & editing

* E-mail: pokoh@gnu.ac.kr

Affiliation Department of Anatomy, College of Veterinary Medicine, Research Institute of Life Science, Gyeongsang National University, Jinju, South Korea

https://orcid.org/0000-0003-0091-8287

⨯

Retinoic acid alleviates the reduction of Akt and Bad phosphorylation and regulates Bcl-2 family protein interactions in animal models of ischemic stroke

Ju-Bin Kang,
Phil-Ok Koh

Published: May 16, 2024
https://doi.org/10.1371/journal.pone.0303213

About the Authors

Ju-Bin Kang

Roles Data curation, Methodology, Software, Visualization, Writing – original draft

Affiliation Department of Anatomy, College of Veterinary Medicine, Research Institute of Life Science, Gyeongsang National University, Jinju, South Korea

Phil-Ok Koh

Roles Conceptualization, Funding acquisition, Supervision, Writing – original draft, Writing – review & editing

* E-mail: pokoh@gnu.ac.kr

Affiliation Department of Anatomy, College of Veterinary Medicine, Research Institute of Life Science, Gyeongsang National University, Jinju, South Korea

https://orcid.org/0000-0003-0091-8287

Competing Interests

The authors have declared that no competing interests exist.

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Retinoic acid
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Ischemic stroke
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Apoptosis
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Neuroprotectives
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Apoptotic signaling cascade
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Brain damage
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Cerebral cortex
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Retinoic acid signaling cascade
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