The Role of Atypical Protein Kinase C in CSF-1-Dependent Erk Activation and Proliferation in Myeloid Progenitors and Macrophages

Angel W. Lee

doi:10.1371/journal.pone.0025580

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Open Access

Peer-reviewed

Research Article

The Role of Atypical Protein Kinase C in CSF-1-Dependent Erk Activation and Proliferation in Myeloid Progenitors and Macrophages

Angel W. Lee

* E-mail: awmlee@angelleelab.org

Affiliations Department of Pharmacology, The University of Michigan Medical School, Ann Arbor, Michigan, United States of America, Center for Proteomics and Systems Biology, The Institute of Molecular Medicine at The University of Texas Health Sciences Center, Houston, Texas, United States of America, OncProTech, Ann Arbor, Michigan, United States of America
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The Role of Atypical Protein Kinase C in CSF-1-Dependent Erk Activation and Proliferation in Myeloid Progenitors and Macrophages

Angel W. Lee

Published: October 18, 2011
https://doi.org/10.1371/journal.pone.0025580

About the Authors

Angel W. Lee

* E-mail: awmlee@angelleelab.org

Affiliations Department of Pharmacology, The University of Michigan Medical School, Ann Arbor, Michigan, United States of America, Center for Proteomics and Systems Biology, The Institute of Molecular Medicine at The University of Texas Health Sciences Center, Houston, Texas, United States of America, OncProTech, Ann Arbor, Michigan, United States of America

Competing Interests

The author has declared that no competing interests exist.

Author Contributions

Conceived and designed the experiments: AWL. Performed the experiments: AWL. Analyzed the data: AWL. Wrote the paper: AWL.

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Phosphorylation
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Immunoprecipitation
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Macrophages
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Transcription factors
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Transfection
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In vitro kinase assay
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MTS assay
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Cell differentiation
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