Integration in oncogenes plays only a minor role in determining the in vivo distribution of HIV integration sites before or during suppressive antiretroviral therapy
Fig 5
HIV IS in other genes in which proviruses can give the host cell a selective advantage.
Maps were generated for each of the genes in Table 3 as described in Fig 4. For each pair of panels, the map at the top shows the IS distribution for the PBMC infected in vitro, and the on-ART distribution is at the bottom A. STAT5B (309 bp/bin). B. BACH2 (1.48 kb/bin). C. MKL1 (906 bp/bin). D. MYB (151 bp/bin). E. IL2RB (197 bp/bin) F. POU2F1 (828 kb/bin).