(A) Transmission electron micrographs of cardiac sections from SIRT3-KO mice and WT controls six weeks after sham or TAC. (×20,000). (B) Heart extracts from SIRT3-KO mice and WT controls were analyzed for triglyceride and cholesterol esters. (C) Palmitate oxidation rates in perfused hearts from WT and SIRT3-KO mice after sham or TAC. The data are presented as the means ± SEM of three independent experiments. *P<0.05,**P<0.01.
Citation: Chen T, Liu J, Li N, Wang S, Liu H, Li J, et al. (2016) Correction: Mouse SIRT3 Attenuates Hypertrophy-Related Lipid Accumulation in the Heart through the Deacetylation of LCAD. PLoS ONE 11(5): e0155173. https://doi.org/10.1371/journal.pone.0155173
Published: May 4, 2016
Copyright: © 2016 Chen et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.