Fig 1.
a. Epicardial adipose tissue thickness was measured by calculating the maximum thickness perpendicular to the surface of the heart, from the myocardium to the visceral pericardium, at a single axial section level—specifically at the atrioventricular groove at the mid-third of the right coronary artery (RCA) and adjacent to the left anterior descending coronary artery (LAD). B. Umbilical fat tissue thickness was measured at umbilicus level perpendicular to rectus muscle anteriorly.
Fig 2.
a. Hepatic steatosis was assessed by measuring liver attenuation values (in Hounsfield Units, HU) on CT scans using regions of interest (ROIs) drawn within the liver parenchyma. b and c. On dual-echo sequences, the out-of-phase image (c) demonstrates a signal reduction relative to the in-phase image (b).
Fig 3.
Pancreatic fat content was also assessed on CT by measuring attenuation values in the head (a), body (b), and tail (c) regions of the pancreas.
Table 1.
Comparison of EAT and subcutaneous fat thickness across key metabolic and clinical variables.
Table 2.
Univariate and multivariate logistic regression analysis for predictors of diabetes mellitus.
Table 3.
Correlation of EAT thickness with age and pancreatic density measurements.
Table 4.
Univariate and multivariate logistic regression analysis of factors associated with pancreatitis history.
Table 5.
Correlation analysis between umbilical subcutaneous fat thickness and EAT measurements.