Table 1.
Incidences of AML and HCC as a function of dose and radiation quality.
Figure 1.
Incidence (%) of AML following exposure to 300 MeV/n 28Si (▾); 600 MeV/n 56Fe (•); 137Cs gamma rays (o); or 1972SPE protons (♦).
The dose response over the 0-3 Gy dose range (top). The dose response over the 0–0.4 Gy dose range (bottom).
Table 2.
Slopes determined using weighted linear regression for the induction of AML or HCC as a function of radiation qualitya.
Table 3.
RBE values (± S.D) for 300 MeV/n 28Si, 600 MeV/n 56Fe, and 1972SPE protons.
Figure 2.
Incidence (%) of HCC following exposure to 300 MeV/n 28Si (▾); 600 MeV/n 56Fe (•); 137Cs gamma rays (o); or 1972SPE protons (♦).
The dose response over the 0–3 Gy dose range (top). The dose response over the 0–0.4 Gy dose range (bottom).
Figure 3.
Incidence (%) of HCC with metastases to the lung as a function of radiation type.
Figure 4.
The probability for development of HCC corrected for background (solid line) with Abbott's correction [34] and the probability of a single traversal of a 50 µm2 hepatocyte nucleus (dashed line) with a 300 MeV/n 28Si ion (A) or 600 MeV/n 56Fe ion (B).