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Table 1.

Full executable search string by database.

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Fig 1.

Flow diagram of study selection.

A PRISMA-compliant flow chart documenting the identification, screening, and inclusion process. From 11,583 initial citations, 174 met criteria via database search, with an additional 221 identified through country-specific purposive searching, resulting in a total of 395 included studies.

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Table 2.

Characteristics of included studies (N = 395).

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Table 3.

Estimates of the Global burden of HEV-Caused Cases and Deaths for data coverage years.

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Table 4.

Serological and epidemiological data sources informing IHME’s 2021 Hepatitis E prevalence estimates.

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Table 5.

Epidemiology of Hepatitis E virus in WHO South-East Asia Region.

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Table 6.

Epidemiology of Hepatitis E virus in WHO Western Pacific Region.

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Table 7.

Epidemiology of Hepatitis E virus in WHO Eastern Mediterranean Region.

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Table 8.

Epidemiology of Hepatitis E virus in WHO African Region.

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Table 9.

Epidemiology of Hepatitis E virus in WHO Europe Region.

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Table 10.

Epidemiology of Hepatitis E virus in WHO Americas Region.

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Fig 2.

Thematic synthesis of reported risk factors (N = 395).

A conceptual mapping of identified drivers for HEV infection, categorized into environmental sanitation, zoonotic pathways, and socio-cultural practices.

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Fig 3.

Global HEV evidence maturity by WHO region.

A visualization of data “maturity” based on the availability of routine surveillance, diagnostic capacity, and peer-reviewed literature density. It highlights the disparity between high-data regions and those with fragmented epidemiological records.

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Fig 4.

Global discordance: disease burden vs. evidence maturity.

A comparative analysis illustrating the “Paradox of Data,” where regions with the highest estimated HEV incidence often coincide with the lowest levels of evidence maturity and diagnostic infrastructure.

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Fig 5.

Regional virological blind spots and transmission profiles.

Map identifying specific geographic areas lacking genotype-specific (molecular) data. It differentiates regions dominated by waterborne Genotypes 1 and 2 from those primarily characterized by zoonotic Genotypes 3 and 4.

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