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Open Access
Peer-reviewed
Research Article
Coronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease risk
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Dharini Iyer ,
Contributed equally to this work with: Dharini Iyer, Quanyi Zhao
Roles Conceptualization, Formal analysis, Investigation, Validation
Affiliation Department of Medicine and Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, United States of America
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Quanyi Zhao ,
Contributed equally to this work with: Dharini Iyer, Quanyi Zhao
Roles Formal analysis, Investigation
Affiliation Department of Medicine and Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, United States of America
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Robert Wirka,
Roles Formal analysis, Methodology
Affiliation Department of Medicine and Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, United States of America
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Ameay Naravane,
Roles Investigation
Affiliation Department of Medicine and Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, United States of America
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Trieu Nguyen,
Roles Investigation, Methodology
Affiliation Department of Medicine and Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, United States of America
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Boxiang Liu,
Roles Investigation, Methodology
Affiliation Department of Medicine and Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, United States of America
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Manabu Nagao,
Roles Investigation, Methodology
Affiliation Department of Medicine and Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, United States of America
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Paul Cheng,
Roles Conceptualization, Formal analysis
Affiliation Department of Medicine and Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, United States of America
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Clint L. Miller,
Roles Conceptualization, Formal analysis, Investigation
Affiliation Departments of Public Health Sciences, Biochemistry and Genetics, and Biomedical Engineering, University of Virginia, Charlottesville, VA, United States of America
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Juyong Brian Kim,
Roles Conceptualization, Investigation, Methodology
Affiliation Department of Medicine and Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, United States of America
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Milos Pjanic,
Roles Data curation, Software
Affiliation Department of Medicine and Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, United States of America
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Thomas Quertermous
Roles Conceptualization, Funding acquisition, Investigation, Project administration, Resources, Supervision, Writing – original draft, Writing – review & editing
* E-mail: tomq1@stanford.edu
Affiliation Department of Medicine and Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, United States of America
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Coronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease risk
- Dharini Iyer,
- Quanyi Zhao,
- Robert Wirka,
- Ameay Naravane,
- Trieu Nguyen,
- Boxiang Liu,
- Manabu Nagao,
- Paul Cheng,
- Clint L. Miller,
- Juyong Brian Kim
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- Published: October 11, 2018
- https://doi.org/10.1371/journal.pgen.1007681