Advertisement

  • Loading metrics

Open Access

Correction

Correction: IFNγ Signaling Endows DCs with the Capacity to Control Type I Inflammation during Parasitic Infection through Promoting T-bet+ Regulatory T Cells

  • The PLOS Pathogens Staff
  • Article
  • Metrics
  • Comments
  • Media Coverage

There are errors in the Funding section. The correct funding information is as follows: This work was supported by NIH grants AI089935 and AI108651 (L.-F.L). L.-F.L. is a Kimmel Scholar and a Hellman Fellow. H.-M.L is an Irvington Fellow of the Cancer Research Institute. The conditional IFNγR2 mutant mouse generation was supported by DFG (SFB 621 to W.M.) and MUGEN LSHG-CT-2005-005203. The initial functional characterization of IFNγR2 mutant mice was supported by the BBSRC PhD program at the University of Manchester. The aforementioned funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.

Reference

  1. 1. Lee H-M, Fleige A, Forman R, Cho S, Khan AA, Lin L-L, et al. (2015) IFNγ Signaling Endows DCs with the Capacity to Control Type I Inflammation during Parasitic Infection through Promoting T-bet+ Regulatory T Cells. PLoS Pathog 11(2): e1004635. pmid:25658840

Citation: The PLOS Pathogens Staff (2015) Correction: IFNγ Signaling Endows DCs with the Capacity to Control Type I Inflammation during Parasitic Infection through Promoting T-bet+ Regulatory T Cells. PLoS Pathog 11(3): e1004741. https://doi.org/10.1371/journal.ppat.1004741

Published: March 16, 2015

Copyright: © 2015 The PLOS Pathogens Staff. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited