Advertisement
  • Loading metrics

Rhinovirus Attenuates Non-typeable Hemophilus influenzae-stimulated IL-8 Responses via TLR2-dependent Degradation of IRAK-1

  • Benjamin L. Unger ,

    Contributed equally to this work with: Benjamin L. Unger, Andrea N. Faris

    Affiliation Department of Pediatrics and Communicable Diseases, University of Michigan, Ann Arbor, Michigan, United States of America

  • Andrea N. Faris ,

    Contributed equally to this work with: Benjamin L. Unger, Andrea N. Faris

    Affiliation Department of Pediatrics and Communicable Diseases, University of Michigan, Ann Arbor, Michigan, United States of America

  • Shyamala Ganesan,

    Affiliation Department of Pediatrics and Communicable Diseases, University of Michigan, Ann Arbor, Michigan, United States of America

  • Adam T. Comstock,

    Affiliation Department of Pediatrics and Communicable Diseases, University of Michigan, Ann Arbor, Michigan, United States of America

  • Marc B. Hershenson,

    Affiliations Department of Pediatrics and Communicable Diseases, University of Michigan, Ann Arbor, Michigan, United States of America, Department of Molecular and Integrative Physiology, University of Michigan, Ann Arbor, Michigan, United States of America

  • Umadevi S. Sajjan

    usajjan@umich.edu

    Affiliation Department of Pediatrics and Communicable Diseases, University of Michigan, Ann Arbor, Michigan, United States of America

Rhinovirus Attenuates Non-typeable Hemophilus influenzae-stimulated IL-8 Responses via TLR2-dependent Degradation of IRAK-1

  • Benjamin L. Unger, 
  • Andrea N. Faris, 
  • Shyamala Ganesan, 
  • Adam T. Comstock, 
  • Marc B. Hershenson, 
  • Umadevi S. Sajjan
PLOS
x

Viewed info

Cited info

Saved info

Discussed info

Questions or concerns about usage data? Please let us know.