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Mycobacterium tuberculosis WhiB3 Maintains Redox Homeostasis by Regulating Virulence Lipid Anabolism to Modulate Macrophage Response

  • Amit Singh,

    Affiliation Department of Microbiology, Centers for Free Radical Biology, AIDS Research, and Emerging Infections and Emergency Preparedness, University of Alabama at Birmingham, Birmingham, Alabama, United States of America

  • David K. Crossman,

    Affiliation Department of Microbiology, Centers for Free Radical Biology, AIDS Research, and Emerging Infections and Emergency Preparedness, University of Alabama at Birmingham, Birmingham, Alabama, United States of America

  • Deborah Mai,

    Affiliation Department of Microbiology, Centers for Free Radical Biology, AIDS Research, and Emerging Infections and Emergency Preparedness, University of Alabama at Birmingham, Birmingham, Alabama, United States of America

  • Loni Guidry,

    Affiliation Department of Microbiology, Centers for Free Radical Biology, AIDS Research, and Emerging Infections and Emergency Preparedness, University of Alabama at Birmingham, Birmingham, Alabama, United States of America

  • Martin I. Voskuil,

    Affiliation Department of Microbiology, University of Colorado Denver, School of Medicine, Aurora, Colorado, United States of America

  • Matthew B. Renfrow,

    Affiliation Department of Biochemistry and Molecular Genetics, University of Alabama at Birmingham, Birmingham, Alabama, United States of America

  • Adrie J. C. Steyn

    asteyn@uab.edu

    Affiliation Department of Microbiology, Centers for Free Radical Biology, AIDS Research, and Emerging Infections and Emergency Preparedness, University of Alabama at Birmingham, Birmingham, Alabama, United States of America

Mycobacterium tuberculosis WhiB3 Maintains Redox Homeostasis by Regulating Virulence Lipid Anabolism to Modulate Macrophage Response

  • Amit Singh, 
  • David K. Crossman, 
  • Deborah Mai, 
  • Loni Guidry, 
  • Martin I. Voskuil, 
  • Matthew B. Renfrow, 
  • Adrie J. C. Steyn
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