Advertisement
Browse Subject Areas
?

Click through the PLOS taxonomy to find articles in your field.

For more information about PLOS Subject Areas, click here.

  • Loading metrics

Open Access

Correction

Correction: Human alpha defensin 5 is a candidate biomarker to delineate inflammatory bowel disease

  • Amanda D. Williams,
  • Olga Y. Korolkova,
  • Amos M. Sakwe,
  • Timothy M. Geiger,
  • Samuel D. James,
  • Roberta L. Muldoon,
  • Alan J. Herline,
  • J. Shawn Goodwin,
  • Michael G. Izban,
  • Mary K. Washington,
  • Duane T. Smoot,
  • Billy R. Ballard,
  • Maria Gazouli,
  • Amosy E. M’Koma
  • Article
  • Metrics
  • Comments
  • Media Coverage

In Fig 6, the “Active Disease” image for WD- 12919 in Fig 6A was incorrectly duplicated in Fig 6C. Please see the correct Fig 6 here.

thumbnail
Download:
Fig 6. Assessment of HD5 and Paneth cells in inflamed and normal, adjacent tissue.

HD5 staining of CC inflamed and normal, adjacent tissue shows expression of HD5 in all patient samples examined (Fig. 6A), compared to inflamed and adjacent, normal tissue of UC patients (Fig. 6B). H&E stains for Paneth Cells (Fig. 6C and D), were negative for PCs in all tissues.

https://doi.org/10.1371/journal.pone.0189551.g001

Reference

  1. 1. Williams AD, Korolkova OY, Sakwe AM, Geiger TM, James SD, Muldoon RL, et al. (2017) Human alpha defensin 5 is a candidate biomarker to delineate inflammatory bowel disease. PLoS ONE 12(8): e0179710. https://doi.org/10.1371/journal.pone.0179710 pmid:28817680

Citation: Williams AD, Korolkova OY, Sakwe AM, Geiger TM, James SD, Muldoon RL, et al. (2017) Correction: Human alpha defensin 5 is a candidate biomarker to delineate inflammatory bowel disease. PLoS ONE 12(12): e0189551. https://doi.org/10.1371/journal.pone.0189551

Published: December 6, 2017

Copyright: © 2017 Williams et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.