The Acknowledgments section is incomplete. The correct Acknowledgments should appear as follows: We thank Aaron Darling and Hatch Stokes for their comments on an early draft of this manuscript.
A: The 3´-CS of class 1 integrons in pASL01a and pAKU_1 has been structurally modified by different IS26-mediated deletion events such that PCR with L1 and JL-D2 primers is expected to generate 2082 and 2342 bp long amplicons respectively. B: Eight base pair signature sequences created by the insertion of IS26 found flanking the inverted repeats of IS26 elements clearly suggest that the CRL in strain 2011EC-3493 is a derivative of Tn6029C while 2009–2050 is a derivative of Tn6029B described previously in pHCM1. The eight base repeat on the left end of Tn6029B and that in 2009EL-2050 are different because in pHCM1 (Tn6029B) there is a deletion of the class 1 integron whereas in 2009EL-2050 the fragment adjacent to the left hand end of the insertion site is inverted by IS26-mediated events. These signatures are also consistent with the proposition that Tn6029C evolved from Tn6029B.
Citation: The PLOS ONE Staff (2015) Correction: A Role for Tn6029 in the Evolution of the Complex Antibiotic Resistance Gene Loci in Genomic Island 3 in Enteroaggregative Hemorrhagic Escherichia coli O104:H4. PLoS ONE 10(4): e0126197. doi:10.1371/journal.pone.0126197
Published: April 17, 2015
Copyright: © 2015 The PLOS ONE Staff. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited