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Correction

Correction: Maintenance of Hypertensive Hemodynamics Does Not Depend on ROS in Established Experimental Chronic Kidney Disease

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An error occurred while the manuscript was being prepared for publication. Figures 2 and 3 do not appear in color in the PDF version of the paper, making the images difficult to read.

Please see the corrected Figure 2 here.

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Figure 2. Bilateral ablation (C and F) induced more glomerulosclerosis (panel A) and tubulo-interstitial damage (panel D) in CKD rats compared to controls (B and E) on PAS-stained sections.

Means ± SEM. Unpaired t-test: ###P<0.001 vs. CON.

https://doi.org/10.1371/journal.pone.0088596.g002

Please see the corrected Figure 3 here.

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Figure 3. Less (RECA)+ pixels (green) were found in CKD rats compared to CON rats in both glomeruli (panel A) and tubular fields (panel D).

Immunohistochemical labeling is shown in CON rats (panels B and E) and in CKD rats (panels C and F). Means ± SEM. Unpaired t-test: ###P<0.001; ##P<0.01 vs. CON.

https://doi.org/10.1371/journal.pone.0088596.g003

Reference

  1. 1. Papazova DA, van Koppen A, Koeners MP, Bleys RL, Verhaar MC, et al. (2014) Maintenance of Hypertensive Hemodynamics Does Not Depend on ROS in Established Experimental Chronic Kidney Disease. PLoS ONE 9(2): e88596

Citation: The PLOS ONE Staff (2014) Correction: Maintenance of Hypertensive Hemodynamics Does Not Depend on ROS in Established Experimental Chronic Kidney Disease. PLoS ONE 9(6): e99476. https://doi.org/10.1371/journal.pone.0099476

Published: June 4, 2014

Copyright: © 2014 The PLOS ONE Staff. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.