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Open Access
Peer-reviewed
Research Article
HA117 endows HL60 cells with a stem-like signature by inhibiting the degradation of DNMT1 via its ability to down-regulate expression of the GGL domain of RGS6
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Shuangshuang Li,
Affiliations Department of Gastrointestinal Surgery and Neonatal Surgery, Children's Hospital of Chongqing Medical University, Ministry of Education Key Laboratory of Child Development and Disorders, Chongqing, China, China International Science and Technology Cooperation base of Child Development and Critical Disorders, Chongqing Medical University, Chongqing, China, Chongqing Key Laboratory of Pediatrics, Chongqing Medical University, Chongqing, China, Department of Pediatrics, Chongqing Medical University, Chongqing, China
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Xianqing Jin ,
* E-mail: etzhl@163.com
Affiliations Department of Gastrointestinal Surgery and Neonatal Surgery, Children's Hospital of Chongqing Medical University, Ministry of Education Key Laboratory of Child Development and Disorders, Chongqing, China, China International Science and Technology Cooperation base of Child Development and Critical Disorders, Chongqing Medical University, Chongqing, China, Chongqing Key Laboratory of Pediatrics, Chongqing Medical University, Chongqing, China, Department of Pediatrics, Chongqing Medical University, Chongqing, China
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Huan Wu,
Affiliations Department of Gastrointestinal Surgery and Neonatal Surgery, Children's Hospital of Chongqing Medical University, Ministry of Education Key Laboratory of Child Development and Disorders, Chongqing, China, China International Science and Technology Cooperation base of Child Development and Critical Disorders, Chongqing Medical University, Chongqing, China, Chongqing Key Laboratory of Pediatrics, Chongqing Medical University, Chongqing, China, Department of Pediatrics, Chongqing Medical University, Chongqing, China
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Yi Wang,
Affiliations Department of Gastrointestinal Surgery and Neonatal Surgery, Children's Hospital of Chongqing Medical University, Ministry of Education Key Laboratory of Child Development and Disorders, Chongqing, China, China International Science and Technology Cooperation base of Child Development and Critical Disorders, Chongqing Medical University, Chongqing, China, Chongqing Key Laboratory of Pediatrics, Chongqing Medical University, Chongqing, China, Department of Pediatrics, Chongqing Medical University, Chongqing, China
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Xiaoqing Li,
Affiliations Department of Gastrointestinal Surgery and Neonatal Surgery, Children's Hospital of Chongqing Medical University, Ministry of Education Key Laboratory of Child Development and Disorders, Chongqing, China, China International Science and Technology Cooperation base of Child Development and Critical Disorders, Chongqing Medical University, Chongqing, China, Chongqing Key Laboratory of Pediatrics, Chongqing Medical University, Chongqing, China, Department of Pediatrics, Chongqing Medical University, Chongqing, China
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Yuxia Guo,
Affiliations Department of Gastrointestinal Surgery and Neonatal Surgery, Children's Hospital of Chongqing Medical University, Ministry of Education Key Laboratory of Child Development and Disorders, Chongqing, China, China International Science and Technology Cooperation base of Child Development and Critical Disorders, Chongqing Medical University, Chongqing, China, Chongqing Key Laboratory of Pediatrics, Chongqing Medical University, Chongqing, China, Department of Pediatrics, Chongqing Medical University, Chongqing, China
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Shaoyan Liang
Affiliations Department of Gastrointestinal Surgery and Neonatal Surgery, Children's Hospital of Chongqing Medical University, Ministry of Education Key Laboratory of Child Development and Disorders, Chongqing, China, China International Science and Technology Cooperation base of Child Development and Critical Disorders, Chongqing Medical University, Chongqing, China, Chongqing Key Laboratory of Pediatrics, Chongqing Medical University, Chongqing, China, Department of Pediatrics, Chongqing Medical University, Chongqing, China
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HA117 endows HL60 cells with a stem-like signature by inhibiting the degradation of DNMT1 via its ability to down-regulate expression of the GGL domain of RGS6
- Shuangshuang Li,
- Xianqing Jin,
- Huan Wu,
- Yi Wang,
- Xiaoqing Li,
- Yuxia Guo,
- Shaoyan Liang
- Published: June 30, 2017
- https://doi.org/10.1371/journal.pone.0180142